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This makes Ostarine one of the highest yielding SARMs in terms of delivering lean muscleprotein into the muscle.The two groups of athletes used the same amount of Ostarine, 100 g (3.8 ounces) and 95 g (2.4 ounces). On balance, the group with the lower carbohydrate intake had a greater total muscle protein synthesis (TPMS) at one hour, as measured by phosphorylation, than did the group with a higher carbohydrate diet . The effect of different levels of weight loss was not found to be correlated with TPMS or the reduction in serum free leucine and total fat . However, the study was unable to determine the protein contribution to total protein synthesis in the group with the lowest fat intake.Weight loss of >5% of body weight or 3.5% per month was used to define the group being used as "low-maintenance" . These are not the weights used in the study by Darnell et al. .The study by Darnell et al  used a group of athletes who trained hard (2 months per week), had been prescribed a very low-fat diet for 7 months, and had maintained their total daily energy expenditure (DME) by dieting approximately 15-20% more than normal in order to increase muscle protein synthesis (MPS) as much as possible . It is not surprising that the group with a high fat intake and decreased carbohydrate content of ~40% were more responsive to this dietary treatment. In the present study, the effect of a high-carbohydrate/low-fat diet on a group of athletes with an average training volume of 20.5 kg (4.2% body mass) was measured by the addition of a 4-y-old, low-carbohydrate diet. Ostarine was not administered . The current study was designed to see if the carbohydrate content and insulin sensitivity could be affected on the basis of total dietary protein intake in a low-muscle mass population.There are many unanswered questions that might contribute to the discrepant results in these different studies. These questions include:1. What is the difference between the insulin and E 2 responses to a high protein diet and the insulin and E 2 responses to low protein diets in other studies in other population groups? How do their kinetics relate to a change in protein quality? As mentioned, the E 2 responses of E 2-deficient humans and animals to diets with different protein content were not different .2Related Article: